Nokea
Glucocorticoid effects of carbohydrate metabolism is facilitated with:
- A. Gluconeogenesis
- B. Hypoglicemia
- C. Enhance of glucose utilization with peripherial tissues
- D. Decrease of glycogen reserve in the liver
Correct Answer: A
Rationale: Glucocorticoids promote gluconeogenesis, increasing blood glucose levels by converting non-carbohydrate sources into glucose.
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All of the following are adverse effects of phenothiazines except
- A. Rigidity and tremor
- B. Menstrual irregularities
- C. Cholestatic jaundice
- D. Systolic hypertension
Correct Answer: D
Rationale: Phenothiazines cause hypotension, not hypertension.
Which mode of action has insulin in fatty cells?
- A. Decrease glycerol synthesis
- B. Increase synthesis of triglycerides
- C. Decrease fatty acid synthesis
Correct Answer: B
Rationale: Insulin promotes triglyceride synthesis in fat cells by enhancing glucose uptake and lipogenesis.
Which of the following glucocorticosteroids are used for cerebral oedema:
- A. Triamcinolon
- B. Prednisolon
- C. Prednison
- D. Dexamethasone
Correct Answer: D
Rationale: Dexamethasone is preferred for cerebral edema due to its potent anti-inflammatory effects and minimal mineralocorticoid activity.
A patient is being treated with isoniazid (INH), rifampin, and pyrazinamide in phase I of treatment for tuberculosis. The organism develops resistance to isoniazid. Which drug will the nurse anticipate the provider will order to replace the isoniazid?
- A. Ciprofloxacin (Cipro)
- B. Ethambutol (Myambutol)
- C. Kanamycin
- D. Streptomycin sulfate
Correct Answer: B
Rationale: Ethambutol replaces INH in phase I if resistance occurs; ciprofloxacin or kanamycin are less common first-line options. If there is bacterial resistance to isoniazid, the first phase may be changed to ethambutol, rifampin, and pyrazinamide.
Cholinergic blockers are used to:
- A. Counteract the extrapyramidal symptoms (EPS) effects of phenothiazines
- B. Control tremors and relax smooth muscle in Parkinson's disease
- C. Inhibit the muscarinic action of ACh on bladder muscle
- D. All of the above
Correct Answer: D
Rationale: Cholinergic blockers address EPS, Parkinson's symptoms, and bladder overactivity.