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Triglycerides are absorbed in the intestines after a meal. Question: After being absorbed by intestinal cells, triglycerides are transported in the blood circulation by which lipid particle?
- A. Chylomicron particle
- B. HDL particle
- C. IDL particle
- D. VLDL particle
Correct Answer: A
Rationale: Post-meal triglycerides ride chylomicrons gut to blood, not HDL, IDL, or VLDL's liver gig. Nurses track this, a chronic fat ferry.
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People with obesity generally respond more strongly to food cues than non-obese people. Question: Which behavioural training is most indicated to reduce this mechanism?
- A. Cognitive modulation training
- B. Compulsivity training
- C. Extinction training
- D. None of the options above
Correct Answer: C
Rationale: Food cues overdrive extinction dims them, not cognitive tweaks or compulsion drills. Nurses train this, a chronic cue fade.
Mr Soh, a 40-year-old accountant on allopurinol 200 mg OM for the past eight months, reports two recent gout attacks in the past year. He has no other known past medical history. When you probe, he is adherent to allopurinol except for missing it perhaps once or twice a month. His BMI 25 kg/m², BP 144/94 mm Hg. His last uric acid was one month ago, which was 405 mmol/L. He is having a gout attack now. He tells you that his gout attacks are usually aborted with colchicine TDS for two days. Whilst on colchicine, he does not experience diarrhoea except for one episode of loose stools, after which he stops colchicine. Which is the most appropriate next step?
- A. Start Hydrochlorothiazide for hypertension
- B. Start Losartan for hypertension
- C. Stop Allopurinol during this acute gout attack and start colchicine. Consider checking a baseline creatinine if not recently available
- D. Continue allopurinol at 200 mg OM despite the attack and start colchicine. Consider checking an updated uric acid level and creatinine two weeks after the attack resolves. If uric acid is >360, explain that allopurinol 200 mg OM is insufficient and needs to be up titrated
Correct Answer: D
Rationale: Gout mid-attack 405 uric acid on 200 mg allopurinol says it's not enough. Keep it rolling, add colchicine to quash the flare, then recheck labs post-calm to titrate up if >360. Stopping allopurinol spikes urate; HCTZ worsens gout; Losartan's fine but sidesteps; upping now risks confusion. Clinicians stick this path, steering chronic control smart.
Many people with obesity have a lower insulin-stimulated glucose uptake compared with people without increased body weight. It is assumed that several factors play a role in the development of insulin resistance. Question: Which of the following factors is LEAST likely to play a role in the development of insulin resistance?
- A. Reduced insulin levels
- B. Increased adipokine levels
- C. Increased triglyceride levels
- D. Low-grade continuous inflammation
Correct Answer: A
Rationale: Insulin resistance brews from fat's adipokines, triglycerides, inflammation not low insulin, that's type 1's game. Obesity's chronic jam needs excess, not lack nurses flag this misfit.
Which of the following is a treatment option for a client with infective endocarditis?
- A. Bedrest
- B. Antimicrobials
- C. Diet modification
- D. Antihypertensive
Correct Answer: B
Rationale: Infective endocarditis bacterial valve infection needs antimicrobials to kill pathogens like *Streptococcus*, the root cause, halting damage and sepsis. Bedrest aids recovery but doesn't treat. Diet tweaks support health, not infection. Antihypertensives manage pressure, irrelevant to endocarditis's microbial core. Nurses anticipate antibiotics, often IV for weeks, targeting the source, a priority to save valves and lives in this high-mortality condition, aligning with infectious disease protocols.
There are several mechanisms through which AGEs can lead to increased oxidative stress. Question: Which AGE-induced mechanism supposedly plays an important role in the development of oxidative stress in endothelial cells?
- A. Crosslinking of matrix proteins
- B. Induction of apoptosis
- C. Intracellular glycation by methylglyoxal
- D. All three statements provided above are correct
Correct Answer: C
Rationale: AGEs spark stress methylglyoxal's indoor glycation fries cells, outpacing crosslinking or death calls. Key in endothelium, a chronic oxidative kick nurses target this mess.