Which statement is correct regarding glucagon:
- A. Causes hypoglicemia
- B. Causes hyperglicemia
- C. Haven't affect blood glucose level in blood
Correct Answer: B
Rationale: Glucagon causes hyperglycemia by stimulating glycogenolysis and gluconeogenesis in the liver.
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Slow channel syndrome is due to
- A. Accumulation of Na+ ions in channels
- B. Accumulation of K+ ions in channels
- C. Accumulation of Mg++ ions in channels
- D. Accumulation of Ca++ ions in channels
Correct Answer: D
Rationale: Slow channel syndrome relates to calcium channel dysfunction.
AH are prodrugs except
- A. Enalapril
- B. Paracetamol
- C. Sulphamethoxazole
- D. Trimethoprim
Correct Answer: D
Rationale: Enalapril is a prodrug; paracetamol, sulphamethoxazole, and trimethoprim are active drugs.
NSAIDs combat fever by inhibiting which prostaglandin?
- A. Prostaglandin E2
- B. Prostaglandin I2
- C. Prostaglandin D2
- D. Prostaglandin F2
Correct Answer: A
Rationale: Prostaglandin E2 mediates fever in the hypothalamus; NSAIDs reduce fever by inhibiting its synthesis.
Which is true regarding the unit dose drug distribution system?
- A. The inventory is delivered to each nursing unit on a regular and recurring basis.
- B. The system delivers one dose of each medication to be administered until the subsequent delivery of inventory.
- C. The use of single dose packages of drugs dispensed to fill each dose requirement as it is ordered.
- D. The amount of inventory needed to dose all patients on the unit for a 24 hour interval.
Correct Answer: C
Rationale: The unit dose drug distribution system uses single unit packages of drugs dispensed to fill each dose requirement as it is ordered.
Angina is caused by:
- A. Blocking beta1 receptors with a constant binding of noradrenaline
- B. Activating beta2 receptors
- C. Beta1 receptors are not active anymore
- D. Extreme binding of noradrenaline due to up regulation
Correct Answer: D
Rationale: Angina results from myocardial ischemia, often due to excessive demand or reduced supply, which can be exacerbated by upregulated noradrenaline binding increasing heart workload.
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