Nokea
Individualized nutrition therapy for patients using conventional, fixed insulin regimens should include teaching the patient to
- A. eat regular meals at regular times.
- B. restrict calories to promote moderate weight loss.
- C. eliminate sucrose and other simple sugars from the diet.
- D. limit saturated fat intake to 30% of dietary calorie intake.
Correct Answer: A
Rationale: Patients with fixed insulin regimens should eat regular meals to match insulin action and prevent hypoglycemia.
You may also like to solve these questions
The antagonistic hormone to calcitonin is _____.
- A. thyroxine.
- B. growth hormone.
- C. insulin.
- D. parathyroid hormone.
Correct Answer: D
Rationale: The correct answer is D: parathyroid hormone. Calcitonin and parathyroid hormone regulate calcium levels in the blood. Calcitonin lowers blood calcium levels by inhibiting bone resorption, while parathyroid hormone increases blood calcium levels by promoting bone resorption. Thus, they have opposite effects on calcium regulation, making parathyroid hormone the antagonistic hormone to calcitonin. Thyroxine (A), growth hormone (B), and insulin (C) do not directly oppose the action of calcitonin in regulating calcium levels.
Neostigmine is a drug useful in the treatment of myasthenia gravis, but it causes parasympathomimetic effects unless another drug is given first. This drug is:
- A. Atropine
- B. Epinephrine
- C. Isoproterenol
- D. Edrophonium
Correct Answer: A
Rationale: The correct answer is A: Atropine. Neostigmine is a cholinesterase inhibitor that increases acetylcholine levels leading to parasympathomimetic effects. Atropine, an anticholinergic drug, blocks the effects of acetylcholine, preventing excessive stimulation of cholinergic receptors. Administering atropine before neostigmine helps counteract the parasympathomimetic effects, making it a necessary pre-treatment. Epinephrine and isoproterenol are adrenergic drugs and wouldn't counteract the cholinergic effects of neostigmine. Edrophonium is another cholinesterase inhibitor like neostigmine, so it wouldn't be effective in preventing the parasympathomimetic effects.
Which of the following occur due to increased secretion of testosterone during puberty in male humans?
- A. 1, 2, 3 and 4
- B. 1, 2 and 3 only
- C. 1 and 2 only
- D. 3 and 4 only
Correct Answer: A
Rationale: Step 1: Increased muscle growth (1) is due to testosterone promoting protein synthesis, leading to muscle hypertrophy.
Step 2: Deepening of voice (2) is caused by testosterone thickening the vocal cords.
Step 3: Growth of facial hair (3) is stimulated by testosterone promoting hair follicle growth.
Step 4: Enlargement of testes (4) is a direct effect of increased testosterone levels. Therefore, choices A, B, C, and D are analyzed:
- B is incorrect as it excludes the enlargement of testes.
- C is incorrect as it excludes the growth of facial hair.
- D is incorrect as it excludes both muscle growth and the growth of facial hair.
Conclusion: Choice A is correct as it includes all the effects of increased testosterone secretion during puberty in male humans.
Target cells for hypothalamic releasing hormones are in the
- A. thyroid
- B. hypothalamus
- C. anterior pituitary
- D. posterior pituitary
Correct Answer: C
Rationale: The correct answer is C: anterior pituitary. Hypothalamic releasing hormones target cells in the anterior pituitary to stimulate the release of specific pituitary hormones. The hypothalamus produces releasing hormones that travel through the hypophyseal portal system and bind to receptors on cells in the anterior pituitary, triggering the release of hormones such as ACTH, TSH, LH, FSH, and GH. This interaction between the hypothalamus and anterior pituitary forms the hypothalamic-pituitary axis, regulating various physiological processes. Choices A (thyroid) and D (posterior pituitary) are incorrect as they are not the primary targets of hypothalamic releasing hormones. Choice B (hypothalamus) is also incorrect as releasing hormones do not target cells within the hypothalamus itself, but rather the anterior pituitary.
Which of the following is NOT a symptom of Addison’s disease?
- A. Salt craving
- B. Weight gain
- C. Hypoglycemia
- D. Hyperpigmentation
Correct Answer: B
Rationale: The correct answer is B: Weight gain. Addison's disease is characterized by adrenal insufficiency, leading to symptoms such as salt craving, hypoglycemia, and hyperpigmentation due to increased melanin production. Weight loss, not weight gain, is commonly associated with Addison's disease due to decreased cortisol levels. Salt craving is due to sodium loss, hypoglycemia results from insufficient cortisol levels, and hyperpigmentation is caused by elevated ACTH levels stimulating melanin production. Weight gain is not a typical symptom, making it the correct answer.