Based on vaginal examination findings, indicators of abnormal labor are
- A. Bandl’s ring and oedematous vulva
- B. Oedematous cervix and fetal hypoxia
- C. Hot, dry vagina and arrest in descent
- D. Maternal distress and severe moulding
Correct Answer: C
Rationale: A hot, dry vagina and lack of descent suggest abnormal labor patterns.
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The AGACNP is receiving report from the recovery room on a patient who just had surgical resection for pheochromocytoma. He knows that which class of drugs should be available immediately to manage hypertensive crisis, a possible consequence of physical manipulation of the adrenal medulla?
- A. Alpha-adrenergic antagonists
- B. Beta-adrenergic antagonists
- C. Intravenous vasodilators
- D. Arteriolar dilators
Correct Answer: C
Rationale: Surgical manipulation of the adrenal medulla during resection of a pheochromocytoma can result in the release of catecholamines leading to a hypertensive crisis. Alpha-adrenergic antagonists, such as phenoxybenzamine or prazosin, are the drugs of choice for managing hypertensive crisis in this situation. These medications block the alpha-adrenergic receptors, leading to vasodilation and lowering of blood pressure. Beta-adrenergic antagonists are contraindicated in this scenario due to the risk of unopposed alpha-adrenergic stimulation that could worsen the hypertension. Intravenous vasodilators and arteriolar dilators may be used as adjunctive therapy but alpha-adrenergic antagonists are the primary agents for managing hypertensive crisis in this context.
The diagnostic factor of polyhydramnious, based on percussion, is
- A. Muffled fetal heart sounds
- B. Presence of a dull sound
- C. Excessive fetal movements
- D. Presence of a fluid thrill
Correct Answer: D
Rationale: Fluid thrill is a key diagnostic sign of polyhydramnious.
Achalasia is a risk factor for
- A. Squamous cell carcinoma
- B. Gastroesophageal reflux disease
- C. Esophageal atrophy
- D. Malabsorption syndromes
Correct Answer: A
Rationale: Achalasia is a motility disorder characterized by the inability of the lower esophageal sphincter to relax, resulting in difficulty swallowing and impaired movement of food from the esophagus into the stomach. Patients with achalasia have an increased risk of developing squamous cell carcinoma of the esophagus. This risk is due to chronic inflammation, stasis of food in the esophagus, and increased exposure of esophageal mucosa to irritants, all of which can contribute to the development of cancer over time. Gastroesophageal reflux disease (Choice B) is less likely to be associated with achalasia because the impaired esophageal motility in achalasia results in decreased, rather than increased, reflux of stomach contents into the esophagus. Esophageal atrophy (Choice C) and malabsorption syndromes (Choice D) are not directly related to achalasia.
What is the role of folic acid in pregnancy?
- A. Prevents gestational hypertension
- B. Supports placenta growth
- C. Prevents neural tube defects
- D. Reduces anemia
Correct Answer: C
Rationale: Folic acid is crucial for preventing neural tube defects.
The AGACNP knows that which of the following must be evaluated as a cause of her abdominal pain?
- A. HELLP syndrome
- B. Placental abruption
- C. Spontaneous hepatic rupture
- D. Preterm labor
Correct Answer: C
Rationale: Abdominal pain in pregnancy can be caused by various conditions, but spontaneous hepatic rupture is a rare but life-threatening cause that must be ruled out. Spontaneous hepatic rupture, also known as liver rupture, can occur in pregnancy, though it is extremely rare. It is associated with severe abdominal pain, hypovolemic shock, and can lead to significant maternal and fetal morbidity and mortality. Therefore, the AGACNP should evaluate this possibility when assessing a pregnant patient with abdominal pain to provide timely and appropriate management.HELLP syndrome, placental abruption, and preterm labor are important considerations in the differential diagnosis of abdominal pain in pregnancy but are not specific to the life-threatening nature of spontaneous hepatic rupture.