Nokea
Cretinism, myxedema, and Graves’ disease are all:
- A. thyroid hormone deficiency diseases.
- B. thyroid hormone disorders caused by oversecretion of thyroxine.
- C. calcium imbalances due primarily to the lack of calcitonin.
- D. disorders of thyroid gland function.
Correct Answer: D
Rationale: Step 1: Cretinism, myxedema, and Graves' disease are all related to thyroid gland function.
Step 2: Cretinism is a congenital hypothyroidism, myxedema is adult hypothyroidism, and Graves' disease is hyperthyroidism.
Step 3: Thus, they are all disorders of thyroid gland function.
Summary:
- Choice A is incorrect because they are not all thyroid hormone deficiency diseases.
- Choice B is incorrect because they are not all caused by oversecretion of thyroxine.
- Choice C is incorrect because they are not primarily related to calcium imbalances due to lack of calcitonin.
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A patient presents with a bump on his neck. Upon taking a tissue sample, there appears to be parafollicular cells. The tumor is also associated with MEN II, although most of the time it is sporadic. What is the best way to moniter treatment?
- A. Measure Thyroid Hormone Levels
- B. Measure Iodine Levels
- C. Measure Tsh Levels
- D. Measure Calcitonin Levels
Correct Answer: D
Rationale: Rationale: The parafollicular cells in the tissue sample indicate a tumor of the thyroid gland, likely medullary thyroid carcinoma (MTC) associated with Multiple Endocrine Neoplasia Type II (MEN II). MTC primarily secretes calcitonin. Monitoring Calcitonin levels is crucial for assessing disease progression and treatment response in MTC. Measuring Thyroid Hormone Levels (A) would not be specific for MTC. Iodine Levels (B) are not relevant in this context. TSH Levels (C) reflect pituitary feedback and are not specific to MTC. Therefore, the best way to monitor treatment in this case is to measure Calcitonin Levels (D).
Which of the following hormones is released from the posterior pituitary gland?
- A. Antidiuretic hormone
- B. Thyroid-stimulating hormone
- C. Parathyroid hormone
- D. Prolactin
Correct Answer: A
Rationale: Antidiuretic hormone (ADH) is released by the posterior pituitary and plays a key role in regulating water balance in the body.
During care of the patient with SIADH, what should the nurse do?
- A. Monitor neurologic status at least every 2 hours.
- B. Teach the patient receiving treatment with diuretics to restrict sodium intake.
- C. Keep the head of the bed elevated to prevent antidiuretic hormone (ADH) release.
- D. Notify the health care provider if the patient's blood pressure decreases more than 20 mm Hg from baseline.
Correct Answer: A
Rationale: Frequent neurologic monitoring is essential for patients with SIADH due to the risk of cerebral edema from hyponatremia.
Which two elements react together to form an ionic compound?
- A. R and T
- B. T and X
- C. X and Z
- D. Z and R
Correct Answer: C
Rationale: The correct answer is C, X and Z. Ionic compounds are formed when a metal (X) reacts with a nonmetal (Z) by transferring electrons to achieve a stable octet configuration. Metal atoms tend to lose electrons to form positively charged cations, while nonmetal atoms tend to gain electrons to form negatively charged anions. In this case, X (metal) and Z (nonmetal) will react to form an ionic compound through electron transfer. Choices A, B, and D are incorrect because they do not involve a metal-nonmetal combination, which is essential for the formation of an ionic compound.
Your patient is diagnosed with myasthenia gravis, and she is given a prescription for pyridostigmine. Which of the following best summarizes the drug’s mechanism of action?
- A. Blocks muscarinic receptors, so that acetylcholine preferentially stimulates skeletal muscle
- B. Inhibits reuptake of acetylcholine by motor nerves
- C. Inhibits skeletal muscle activation at rest so that the muscles are less susceptible to fatigue during exercise
- D. Inhibits breakdown of acetylcholine, thereby improving activation of skeletal muscle by acetylcholine
Correct Answer: D
Rationale: The correct answer is D: "Inhibits breakdown of acetylcholine, thereby improving activation of skeletal muscle by acetylcholine."
Rationale:
1. Pyridostigmine is an acetylcholinesterase inhibitor, which means it blocks the breakdown of acetylcholine.
2. In myasthenia gravis, there is a decrease in acetylcholine receptor sites, leading to muscle weakness.
3. By inhibiting the breakdown of acetylcholine, pyridostigmine increases the availability of acetylcholine at the neuromuscular junction, improving muscle activation.
4. This mechanism helps alleviate muscle weakness in myasthenia gravis patients.
Summary:
A: Incorrect. Pyridostigmine does not block muscarinic receptors or change the preference of acetylcholine stimulation.
B: Incorrect. Pyridostigmine does not inhibit reuptake of acetylcholine; it acts by inhibiting its breakdown