Nokea
Simple goiter can be prevented by _________.
- A. antibiotics
- B. adding iodine to the diet
- C. surgery
- D. hormone therapy
Correct Answer: B
Rationale: The correct answer is B: adding iodine to the diet. Iodine deficiency is a common cause of simple goiter. Iodine is essential for the production of thyroid hormones, and a lack of it can lead to the enlargement of the thyroid gland. Adding iodine to the diet helps prevent simple goiter by ensuring the thyroid has enough iodine to function properly. Antibiotics (choice A) are not effective for preventing goiter as it is not caused by bacteria. Surgery (choice C) and hormone therapy (choice D) are typically used to treat goiter once it has developed, not prevent it.
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Complete the following sentence: Antidiuretic hormone is secreted from the…
- A. Adrenal cortex
- B. Thyroid gland
- C. Pancreas
- D. Posterior pituitary gland
Correct Answer: D
Rationale: The correct answer is D: Posterior pituitary gland. Antidiuretic hormone (ADH), also known as vasopressin, is produced in the hypothalamus and then transported to the posterior pituitary gland for secretion into the bloodstream. ADH plays a key role in regulating water balance by controlling the reabsorption of water in the kidneys. Choices A, B, and C are incorrect as they are not the primary sites of ADH production and secretion. The adrenal cortex produces hormones like cortisol and aldosterone, the thyroid gland produces hormones like thyroxine, and the pancreas produces insulin and glucagon.
Which one of the following is NOT typical of the changes that follow the binding of a hormone to its target cells:
- A. plasma membrane permeability changes
- B. cellular mutations occur
- C. enzymes are activated or inactivated
- D. mitosis is stimulated
Correct Answer: B
Rationale: The correct answer is B: cellular mutations occur. Hormone binding typically triggers signaling cascades that lead to cellular responses like changing plasma membrane permeability, activating/inactivating enzymes, or stimulating mitosis. Cellular mutations are not a direct result of hormone binding and are typically caused by external factors like radiation or chemicals. In this context, mutations do not happen as an immediate response to hormone binding. Therefore, option B is the correct choice. Plasma membrane permeability changes, enzyme activation/inactivation, and mitosis stimulation are all typical outcomes of hormone binding and subsequent signaling pathways.
Gonadocorticoid(s) ________.
- A. synthesized by the adrenal medulla are primarily androgens
- B. production by the adrenal gland is insignificant compared with sex hormone release from the gonads during late puberty
- C. secretion inhibition is highly dependent on a negative feedback loop involving ACTH
- D. hypersecretion can result in adrenogenital syndrome, also called feminization
Correct Answer: B
Rationale: The correct answer is B because during late puberty, the production of sex hormones by the gonads significantly outweighs the production of gonadocorticoids by the adrenal gland. This is due to the maturation and increased activity of the gonads during this developmental stage.
Rationale:
A: Synthesized by the adrenal medulla are primarily catecholamines, not androgens.
C: Secretion inhibition of gonadocorticoids is primarily regulated by the renin-angiotensin-aldosterone system, not solely by a negative feedback loop involving ACTH.
D: Hypersecretion of gonadocorticoids can lead to conditions like Cushing's syndrome, not adrenogenital syndrome or feminization.
Which hormone is responsible for the development of male secondary sexual characteristics?
- A. Androgens
- B. Follicle-stimulating hormone
- C. Epinephrine
- D. Norepinephrine
Correct Answer: A
Rationale: The correct answer is A: Androgens. Androgens, such as testosterone, are responsible for the development of male secondary sexual characteristics, such as facial hair, deep voice, and muscle growth. Follicle-stimulating hormone (B) is involved in sperm production, not male secondary sexual characteristics. Epinephrine (C) and norepinephrine (D) are stress hormones and not related to the development of male secondary sexual characteristics.
What is the most likely effect of a deficiency of aldosterone?
- A. High blood pressure
- B. Low level of potassium in the blood
- C. Low blood volume and hypotension
- D. Goiter formation
Correct Answer: C
Rationale: Rationale:
1. Aldosterone regulates sodium and water balance in the body.
2. Deficiency leads to decreased sodium reabsorption, causing low blood volume.
3. Low blood volume results in hypotension due to reduced blood flow to tissues.
4. Choices A and B are incorrect as aldosterone deficiency does not cause high blood pressure or low potassium levels.
5. Choice D is incorrect as goiter formation is mainly related to thyroid hormone imbalance, not aldosterone deficiency.