Nokea
The nurse is admitting an oncology patient to the unit prior to surgery. The nurse reads in the electronic health record that the patient has just finished radiation therapy. With knowledge of the consequent health risks, the nurse should prioritize assessments related to what health problem?
- A. Cognitive deficits
- B. Impaired wound healing
- C. Cardiac tamponade
- D. Tumor lysis syndrome
Correct Answer: B
Rationale: Radiation pre-surgery zaps tissue impaired wound healing's the big risk, as it fries skin and vessels, slowing repair post-op. Cognitive deficits need brain radiation, not specified. Tamponade's rare, tied to chest radiation and fluid buildup. TLS hits post-chemo, not pre-surgery. Nurses in oncology lock onto skin checks and infection signs, knowing radiation's legacy can tank surgical outcomes if ignored.
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Glibenclamide belongs to the class
- A. Sulphonylureas
- B. Thiazolidinediones
- C. Benzoic acid derivatives
- D. Biguanides
Correct Answer: A
Rationale: Glibenclamide's a sulphonylurea pumps insulin from beta cells, a classic diabetes fix. Thiazolidinediones tweak sensitivity, benzoic acids like repaglinide hit fast, biguanides like metformin curb liver glucose. It's a chronic pancreas prod, not a sensitivity or liver play nurses and pharmacists peg it for type 2's insulin lag, a distinct class with a clear job.
A nurse is caring for a client diagnosed with atherosclerosis. Which of the following is considered a risk factor for the development of this disorder?
- A. Diet high in vitamin K
- B. Low HDL-C/High LDL-C
- C. High HDL-C/Low LDL-C
- D. Vegan diet
Correct Answer: B
Rationale: Atherosclerosis loves lipids low HDL (good cholesterol) and high LDL (bad cholesterol) pile plaque, a prime risk factor driving vessel narrowing. Vitamin K aids clotting, not plaque. High HDL/low LDL protects. Vegan diets cut fats, lowering risk. Nurses flag lipid imbalance, pushing statins or diet shifts, a cholesterol-fueled root of this vascular scourge.
Risk factors for developing COPD do not include:
- A. Smoking - passive or active
- B. Age
- C. High fat diet
- D. Indoor and outdoor air pollution
Correct Answer: C
Rationale: COPD's lung wreckers smoking, age, pollution scar airways, no dodge. High fat diet fattens, not chokes lungs; it's metabolic, not respiratory. Nurses target smoke and smog, not butter, a chronic breath stealer's true culprits.
Non modifiable risk factors for developing chronic illness include:
- A. Smoking and hypertension
- B. Sedentary lifestyle and diabetes
- C. Family history and socio-political factors
- D. Working/living conditions and stress
Correct Answer: C
Rationale: Non-modifiable risk factors are inherent traits or circumstances that cannot be changed, unlike modifiable factors tied to behavior or environment. Smoking and hypertension are modifiable through lifestyle changes or medical intervention, not fixed. Sedentary lifestyle is a choice, and diabetes, while influenced by genetics, is often manageable, making them modifiable. Family history, such as genetic predisposition to diseases like cancer or heart disease, is unalterable, and socio-political factors like access to healthcare shaped by policy or socioeconomic status are beyond individual control, fitting the non-modifiable category. Working and living conditions, plus stress, can be adjusted with resources or coping strategies, classifying them as modifiable. The distinction lies in control: family history and socio-political factors remain static, influencing chronic illness risk without personal alteration, as noted in foundational chronic disease literature like Farrell (2017), emphasizing genetics and societal context over mutable habits.
The followings are risk factors associated with non-alcoholic fatty liver disease (NAFLD) except:
- A. Elevated uric acid
- B. Elevated blood pressure
- C. Diabetes mellitus
- D. Elevated LDL-cholesterol
Correct Answer: A
Rationale: NAFLD ties to metabolic mess hypertension, diabetes, high LDL, and triglycerides fuel fat's liver pile-up, all in. Uric acid links to gout, not NAFLD's core, despite metabolic overlap. Clinicians eye this quartet, not urate, in chronic liver fat's risk map, a key split.