Excess insulin is most apt to cause:
- A. ketosis.
- B. hypoglycemia.
- C. hypertension.
- D. diuresis.
Correct Answer: B
Rationale: Excess insulin leads to increased glucose uptake by cells, causing a drop in blood sugar levels, resulting in hypoglycemia. This can lead to symptoms like confusion, sweating, and weakness. Ketosis (A) is caused by low insulin levels, hypertension (C) is not directly related to excess insulin, and diuresis (D) is more commonly associated with conditions like diabetes insipidus.
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The antagonistic hormone to aldosterone is ____.
- A. atrial natriuretic hormone (ANH).
- B. antidiuretic hormone (ADH).
- C. cortisol.
- D. glucagon.
Correct Answer: A
Rationale: The correct answer is A: atrial natriuretic hormone (ANH). ANH opposes the actions of aldosterone by promoting salt and water excretion, thus reducing blood volume and blood pressure. ANH is released in response to high blood pressure or high blood volume.
B: antidiuretic hormone (ADH) is not the antagonistic hormone to aldosterone; it acts on the kidneys to regulate water balance.
C: cortisol is a stress hormone and is not directly involved in regulating sodium and water balance.
D: glucagon is a hormone that increases blood sugar levels and is not related to the regulation of aldosterone.
Excess growth hormone would cause all the following EXCEPT __________.
- A. diabetes
- B. suppression of cancer
- C. giantism in children
- D. acromegally in adults
Correct Answer: B
Rationale: Excess growth hormone suppresses cancer by promoting healthy cell growth and inhibiting apoptosis. Diabetes can result from growth hormone excess due to insulin resistance. Giantism in children and acromegaly in adults are both caused by excess growth hormone stimulating excessive skeletal and soft tissue growth. Therefore, the correct answer is B because excess growth hormone does not suppress cancer; it actually promotes cell growth and proliferation.
The posterior pituitary stores and releases .
- A. prolactin and oxytocin
- B. oxytocin and antidiuretic hormone (ADH)
- C. ADH and growth hormone
- D. growth hormone and prolactin
Correct Answer: B
Rationale: The correct answer is B: oxytocin and antidiuretic hormone (ADH).
1. The posterior pituitary stores and releases oxytocin and ADH.
2. Oxytocin is responsible for uterine contractions and milk ejection.
3. ADH regulates water balance by controlling water reabsorption in the kidneys.
Therefore, B is the correct answer.
A, C, and D are incorrect because prolactin, growth hormone, and other hormones are not stored or released by the posterior pituitary.
Peptide hormones function by binding to receptor proteins within the ________. Once the hormone binds to a receptor, it activates ______.
- A. cell: a second messenger
- B. cell: particular genes
- C. plasma membrane: a second messenger
- D. plasma membrane: particular genes
Correct Answer: C
Rationale: The correct answer is C because peptide hormones typically bind to receptor proteins on the plasma membrane. This is because peptide hormones are not able to pass through the cell membrane due to their size and polarity. Once the hormone binds to the receptor on the plasma membrane, it activates a second messenger system within the cell, leading to various cellular responses.
Choice A is incorrect because peptide hormones do not bind directly to intracellular components like second messengers inside the cell. Choice B is incorrect because peptide hormones do not directly activate particular genes within the cell. Choice D is incorrect because peptide hormones do not bind directly to genes on the plasma membrane.
The parathyroid glands maintain adequate levels of blood calcium. This is accomplished through ________.
- A. blocking the action of growth hormone
- B. targeting the bone and activating osteoclasts so that calcium will be released
- C. antagonizing the synthesis of calcitonin
- D. slowing the activity of tissues that require calcium for activity
Correct Answer: B
Rationale: The correct answer is B because the parathyroid glands target the bone and activate osteoclasts to release calcium into the bloodstream. This process helps maintain adequate blood calcium levels. By activating osteoclasts, calcium is released from the bone into the blood, ensuring a balance of calcium levels.
A: Blocking the action of growth hormone is unrelated to the function of parathyroid glands in maintaining blood calcium levels.
C: Antagonizing the synthesis of calcitonin is not the primary mechanism through which parathyroid glands regulate blood calcium levels.
D: Slowing the activity of tissues that require calcium for activity would not help maintain adequate blood calcium levels as it does not address the direct release of calcium from bone by activating osteoclasts.