Nokea
Regarding basal metabolic rate:
- A. it is higher in women
- B. it is determined at rest within 12 hours after the last meal
- C. increases by 18% for each 1°C of fever
- D. is about 40Kcal/m2/h in an average man
Correct Answer: D
Rationale: Basal metabolic rate (BMR) is the energy expended by the body at rest to maintain basic physiological functions. Choice D is correct because it states that BMR is about 40Kcal/m2/h in an average man, which is a commonly accepted value. This is based on the energy needed for essential bodily functions such as breathing, circulation, and cell production. Choices A, B, and C are incorrect because: A) BMR is generally higher in men due to their higher muscle mass; B) BMR is measured after an overnight fast, not within 12 hours after the last meal; C) Fever typically increases BMR by around 7% per 1°C rise, not 18%.
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The home care nurse should intervene to correct a patient whose insulin administration includes
- A. warming a prefilled refrigerated syringe in the hands before administration.
- B. storing syringes prefilled with NPH and regular insulin needle-up in the refrigerator.
- C. placing the insulin bottle currently in use in a small container on the bathroom countertop.
- D. mixing an evening dose of regular insulin with insulin glargine in one syringe for administration.
Correct Answer: D
Rationale: Insulin glargine should never be mixed with other insulins. This could alter its action and effectiveness.
What is the hormone produced by adipose tissue which signals satiety?
- A. melatonin
- B. leptin
- C. thymosins
- D. glucagons
Correct Answer: B
Rationale: Leptin is the correct answer because it is a hormone produced by adipose tissue that signals satiety to the brain, helping regulate energy balance and food intake. Melatonin is a hormone that regulates sleep-wake cycles, not hunger. Thymosins are involved in immune function. Glucagon is a hormone that increases blood sugar levels, not related to satiety.
The pituitary gland is related laterally to:
- A. The cavernous sinus
- B. Hypothalamus
- C. Diaphragma sellae
- D. Optic chiasma
Correct Answer: A
Rationale: Step 1: The pituitary gland is located in the sella turcica of the sphenoid bone.
Step 2: The cavernous sinus is a venous sinus located on each side of the sella turcica.
Step 3: The pituitary gland lies laterally to the cavernous sinus.
Step 4: Therefore, the correct answer is A: The cavernous sinus.
Summary: B is incorrect as the hypothalamus is superior to the pituitary gland. C is incorrect as the diaphragma sellae is a part of the dura mater covering the pituitary gland. D is incorrect as the optic chiasma is located superior to the pituitary gland.
Being lipid soluble, steroids can do all the following EXCEPT:
- A. catalyze cyclic AMP
- B. diffuse through the plasma membranes of target cells
- C. enter the nucleus
- D. activate genes to transcribe mRNA for protein synthesis
Correct Answer: A
Rationale: Step 1: Steroids are lipid-soluble molecules, allowing them to easily diffuse through the plasma membrane of target cells.
Step 2: Once inside the cell, steroids can enter the nucleus to bind to specific receptors and regulate gene expression.
Step 3: Steroids do not catalyze cyclic AMP; this function is typically associated with non-steroid hormones like adrenaline.
Step 4: Therefore, the correct answer is A because steroids do not catalyze cyclic AMP, unlike other hormones.
Ritodrine is a drug indicated for suppressing uterine contractions in premature labor. The drug also causes bronchodilation. It has no vasoconstrictor effects, nor any effects on the size of the pupil of the eye. Which of the following is the most likely classification of this drug?
- A. α-adrenergic agonist
- B. Antimuscarinic drug
- C. β-adrenergic agonist
- D. β-adrenergic blocker
Correct Answer: C
Rationale: The correct answer is C: β-adrenergic agonist. Ritodrine is indicated for suppressing uterine contractions, which is a typical effect of β-adrenergic agonists. Bronchodilation is also a known effect of β-adrenergic agonists. Since the drug has no vasoconstrictor effects and does not affect the size of the pupil of the eye, it is unlikely to be an α-adrenergic agonist or an antimuscarinic drug. Additionally, the fact that ritodrine causes bronchodilation further supports its classification as a β-adrenergic agonist.